From well known conspiracy site, the BBC;

Despite the wide acceptance of the conclusions reached by the Nature Medicine Paper, there are still some scientists who refuse to see it as conclusive proof that the virus wasn’t manipulated in some way.
 
Nikolai Petrovsky, a professor of medicine at Flinders University in Adelaide and Chairman and Research Director of Vaxine, a company leading Australia’s search for a Covid-19 vaccine, believes that laboratory manipulation has to remain under consideration for being behind as a possible explanation for Sars-CoV-2’s “surprising” ability to bind to human cells.
 
And he rejects the argument that if it wasn’t pre-planned by a computer model, it didn’t happen.
 
“In science a lot of things are done on the basis of give-it-a-go, and a lot of things happen completely by accident,” he tells me. “You know, a lot of Nobel prizes have been won on the basis of accidents.”
 
Others refer to Wuhan’s past experiments in which genome segments from different viruses have been stitched together to investigate how spike proteins bind to human cells.
 
And they point out that there is no way of knowing whether or not Wuhan does in fact have, among the hundreds of coronaviruses it has collected, anything closer in genetic make-up to Sars-CoV-2 than RaTG13.
 
What’s more, RaTG13 is itself the subject of some speculation.
 
The BBC has had it confirmed, from the researchers running a respected Chinese database, that it is the same virus as one that the WIV previously referred to as RaBatCov/4991in this paper.
 
There is no explanation for the change of name, but that 2016 paper makes it clear that the virus appears to be a new strain of Sars-type coronavirus.
 
Professor Petrovsky says it would be odd if the WIV, given its interest in such viruses, hadn’t continued to work on it. But there is no more mention of it all until this year, when RaBatCov/4991 re-emerged under its new name RaTG13 as the closest known relative of Sars-CoV-2.

This is one creepy little bug.

Most of us have already absorbed the idea that the coronavirus does some weird and sinister things to the human body that are unlike most other respiratory viruses known to man. But now a new study finds yet another unsettling thing that the virus appears to do to help spread from cell to cell.
 
A new study by an international team led by UC San Francisco finds that cells infected with SARS-CoV-2 quickly begin to grow new arms or dendrites — referred to clinically as filopodia — which are themselves studded with fresh virus particles. These filopodia then seek to reach into and through the walls of neighboring cells, thereby infecting them. And this appears to be a second mode that the virus has for replicating and spreading itself in the body. […]
 
Krogan says that while other viruses — including HIV and the family of viruses that cause smallpox — also use filopedia as mechanisms of spreading infection, the way this virus so rapidly prompts the growth of these tentacles is highly unusual. And the shape of them, branching off the cell and each other like trees, is also apparently strange. Other infectious diseases like HIV don’t cause these kinds of prolific, mutant growths.

It almost sounds like something cooked up in a lab. The good news: it may lead to new treatment options.